Delayed administration of neural stem cells after hypoxia–ischemia reduces sensorimotor deficits, ce...
Delayed administration of neural stem cells after hypoxia–ischemia reduces sensorimotor deficits, cerebral lesion size, and neuroinflammation in neonatal mice
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New York: Nature Publishing Group US
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English
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New York: Nature Publishing Group US
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Background:
Hypoxic–ischemic (HI) encephalopathy causes mortality and severe morbidity in neonates. Treatments with a therapeutic window >6 h are currently not available. Here, we explored whether delayed transplantation of allogenic neural stem cells (NSCs) at 10 d after HI could be a tool to repair HI brain injury and improve behavioral impair...
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Delayed administration of neural stem cells after hypoxia–ischemia reduces sensorimotor deficits, cerebral lesion size, and neuroinflammation in neonatal mice
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TN_cdi_proquest_miscellaneous_1835355765
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https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_proquest_miscellaneous_1835355765
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ISSN
0031-3998
E-ISSN
1530-0447
DOI
10.1038/pr.2016.172
