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Diallyl trisulfide ameliorates myocardial ischemia–reperfusion injury by reducing oxidative stress a...

Diallyl trisulfide ameliorates myocardial ischemia–reperfusion injury by reducing oxidative stress a...

https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_proquest_miscellaneous_1893549644

Diallyl trisulfide ameliorates myocardial ischemia–reperfusion injury by reducing oxidative stress and endoplasmic reticulum stress-mediated apoptosis in type 1 diabetic rats: role of SIRT1 activation

About this item

Full title

Diallyl trisulfide ameliorates myocardial ischemia–reperfusion injury by reducing oxidative stress and endoplasmic reticulum stress-mediated apoptosis in type 1 diabetic rats: role of SIRT1 activation

Publisher

New York: Springer US

Journal title

Apoptosis (London), 2017-07, Vol.22 (7), p.942-954

Language

English

Formats

Publication information

Publisher

New York: Springer US

Subjects

Subjects and topics

More information

Scope and Contents

Contents

Diallyl trisulfide (DATS) protects against apoptosis during myocardial ischemia-reperfusion (MI/R) injury in diabetic state, although the underlying mechanisms remain poorly defined. Previously, we and others demonstrated that silent information regulator 1 (SIRT1) activation inhibited oxidative stress and endoplasmic reticulum (ER) stress during M...

Alternative Titles

Full title

Diallyl trisulfide ameliorates myocardial ischemia–reperfusion injury by reducing oxidative stress and endoplasmic reticulum stress-mediated apoptosis in type 1 diabetic rats: role of SIRT1 activation

Identifiers

Primary Identifiers

Record Identifier

TN_cdi_proquest_miscellaneous_1893549644

Permalink

https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_proquest_miscellaneous_1893549644

Other Identifiers

ISSN

1360-8185

E-ISSN

1573-675X

DOI

10.1007/s10495-017-1378-y

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