Loss of CD73-mediated extracellular adenosine production exacerbates inflammation and abnormal alveo...
Loss of CD73-mediated extracellular adenosine production exacerbates inflammation and abnormal alveolar development in newborn mice exposed to prolonged hyperoxia
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New York: Nature Publishing Group US
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English
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New York: Nature Publishing Group US
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Background
Hyperoxic lung injury is characterized by cellular damage from high oxygen concentrations that lead to an inflammatory response and it disrupts normal alveolarization in the developing newborn lung. Adenosine is a signaling molecule that is generated extracellularly by ecto-5′-nucleotidase (CD73) in response to injury. Extracellular a...
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Full title
Loss of CD73-mediated extracellular adenosine production exacerbates inflammation and abnormal alveolar development in newborn mice exposed to prolonged hyperoxia
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TN_cdi_proquest_miscellaneous_1932161407
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https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_proquest_miscellaneous_1932161407
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ISSN
0031-3998,1530-0447
E-ISSN
1530-0447
DOI
10.1038/pr.2017.176
