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Geniposide Attenuates Post-Ischaemic Neurovascular Damage via GluN2A/AKT/ ERK-Dependent Mechanism

Geniposide Attenuates Post-Ischaemic Neurovascular Damage via GluN2A/AKT/ ERK-Dependent Mechanism

https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_proquest_miscellaneous_1945216758

Geniposide Attenuates Post-Ischaemic Neurovascular Damage via GluN2A/AKT/ ERK-Dependent Mechanism

About this item

Full title

Geniposide Attenuates Post-Ischaemic Neurovascular Damage via GluN2A/AKT/ ERK-Dependent Mechanism

Publisher

Basel, Switzerland: S. Karger AG

Journal title

Cellular physiology and biochemistry, 2017-01, Vol.43 (2), p.705-716

Language

English

Formats

Publication information

Publisher

Basel, Switzerland: S. Karger AG

More information

Scope and Contents

Contents

Background/Aims: Calcium-permeable ionotropic NMDAR-mediated hyperactivity is regarded as the critical factor in modulating the development of ischaemic stroke. Recently, there has been increasing interest in preventing post-stroke neuronal death by focusing on intervening in the function of subpopulations of NMDARs and their downstream signalling....

Alternative Titles

Full title

Geniposide Attenuates Post-Ischaemic Neurovascular Damage via GluN2A/AKT/ ERK-Dependent Mechanism

Identifiers

Primary Identifiers

Record Identifier

TN_cdi_proquest_miscellaneous_1945216758

Permalink

https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_proquest_miscellaneous_1945216758

Other Identifiers

ISSN

1015-8987

E-ISSN

1421-9778

DOI

10.1159/000480657

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