Microglial Cells Depletion Increases Inflammation and Modifies Microglial Phenotypes in an Animal Mo...
Microglial Cells Depletion Increases Inflammation and Modifies Microglial Phenotypes in an Animal Model of Severe Sepsis
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New York: Springer US
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Language
English
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New York: Springer US
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Sepsis-associated encephalopathy is highly prevalent and has impact both in early and late morbidity and mortality. The mechanisms by which sepsis induces brain dysfunction include neuroinflammation, disrupted blood–brain barrier, oxidative stress, and microglial activation, but the cellular and molecular mechanisms involved in these events are not...
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Microglial Cells Depletion Increases Inflammation and Modifies Microglial Phenotypes in an Animal Model of Severe Sepsis
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TN_cdi_proquest_miscellaneous_2215031316
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https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_proquest_miscellaneous_2215031316
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ISSN
0893-7648
E-ISSN
1559-1182
DOI
10.1007/s12035-019-1606-2