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Dietary Selenium Deficiency Facilitated Reduced Stomatin and Phosphatidylserine Externalization, Inc...

Dietary Selenium Deficiency Facilitated Reduced Stomatin and Phosphatidylserine Externalization, Inc...

https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_proquest_miscellaneous_2394884911

Dietary Selenium Deficiency Facilitated Reduced Stomatin and Phosphatidylserine Externalization, Increasing Erythrocyte Osmotic Fragility in Mice

About this item

Full title

Dietary Selenium Deficiency Facilitated Reduced Stomatin and Phosphatidylserine Externalization, Increasing Erythrocyte Osmotic Fragility in Mice

Publisher

New York: Springer US

Journal title

Biological trace element research, 2021-02, Vol.199 (2), p.594-603

Language

English

Formats

Publication information

Publisher

New York: Springer US

More information

Scope and Contents

Contents

Selenium (Se) is an essential trace element that maintains normal physiological functions in organisms. Since the discovery of glutathione peroxidase (GSH-PX), public interest in selenoproteins has gradually increased. Based on previous studies, dietary Se maintains erythrocyte homeostasis through selenoprotein-induced mediation of redox reactions....

Alternative Titles

Full title

Dietary Selenium Deficiency Facilitated Reduced Stomatin and Phosphatidylserine Externalization, Increasing Erythrocyte Osmotic Fragility in Mice

Identifiers

Primary Identifiers

Record Identifier

TN_cdi_proquest_miscellaneous_2394884911

Permalink

https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_proquest_miscellaneous_2394884911

Other Identifiers

ISSN

0163-4984

E-ISSN

1559-0720

DOI

10.1007/s12011-020-02162-3

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