Galectin‐3 enhances atrial remodelling and arrhythmogenesis through CD98 signalling
Galectin‐3 enhances atrial remodelling and arrhythmogenesis through CD98 signalling
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Publisher
England: Wiley Subscription Services, Inc
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Language
English
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Publisher
England: Wiley Subscription Services, Inc
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Scope and Contents
Contents
Aim
Galectin‐3 (Gal‐3) is a biomarker of atrial fibrillation (AF) that mediates atrial inflammation. CD98 is the membrane surface receptor for Gal‐3. Nevertheless, the role of the Gal‐3/CD98 axis in atrial arrhythmogenesis is unclear. In this study, we investigated the effects of Gal‐3/CD98 signalling on atrial pathogenesis.
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Full title
Galectin‐3 enhances atrial remodelling and arrhythmogenesis through CD98 signalling
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TN_cdi_proquest_miscellaneous_2618232149
Permalink
https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_proquest_miscellaneous_2618232149
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ISSN
1748-1708
E-ISSN
1748-1716
DOI
10.1111/apha.13784
