Age-associated B cells contribute to the pathogenesis of rheumatoid arthritis by inducing activation...
Age-associated B cells contribute to the pathogenesis of rheumatoid arthritis by inducing activation of fibroblast-like synoviocytes via TNF-α-mediated ERK1/2 and JAK-STAT1 pathways
About this item
Full title
Author / Creator
Qin, Yi , Cai, Ming-Long , Jin, Hui-Zhi , Huang, Wei , Zhu, Chen , Bozec, Aline , Huang, Jingang and Chen, Zhu
Publisher
England: BMJ Publishing Group Ltd and European League Against Rheumatism
Journal title
Language
English
Formats
Publication information
Publisher
England: BMJ Publishing Group Ltd and European League Against Rheumatism
Subjects
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Scope and Contents
Contents
ObjectivesAge-associated B cells (ABCs) are a recently identified B cell subset, whose expansion has been increasingly linked to the pathogenesis of autoimmune disorders. This study aimed to investigate whether ABCs are involved in the pathogenesis and underlying mechanisms of rheumatoid arthritis (RA).MethodsABCs were assessed in collagen-induced...
Alternative Titles
Full title
Age-associated B cells contribute to the pathogenesis of rheumatoid arthritis by inducing activation of fibroblast-like synoviocytes via TNF-α-mediated ERK1/2 and JAK-STAT1 pathways
Authors, Artists and Contributors
Author / Creator
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Primary Identifiers
Record Identifier
TN_cdi_proquest_miscellaneous_2681814146
Permalink
https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_proquest_miscellaneous_2681814146
Other Identifiers
ISSN
0003-4967
E-ISSN
1468-2060
DOI
10.1136/ard-2022-222605
