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Possible Involvement of Advanced Glycation End-Products (AGEs) in the Pathogenesis of Alzheimers Dis...

Possible Involvement of Advanced Glycation End-Products (AGEs) in the Pathogenesis of Alzheimers Dis...

https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_proquest_miscellaneous_69206240

Possible Involvement of Advanced Glycation End-Products (AGEs) in the Pathogenesis of Alzheimers Disease

About this item

Full title

Possible Involvement of Advanced Glycation End-Products (AGEs) in the Pathogenesis of Alzheimers Disease

Publisher

United Arab Emirates: Bentham Science Publishers Ltd

Journal title

Current pharmaceutical design, 2008-04, Vol.14 (10), p.973-978

Language

English

Formats

Publication information

Publisher

United Arab Emirates: Bentham Science Publishers Ltd

More information

Scope and Contents

Contents

Alzheimers disease (AD) is the most common cause of dementia in developed countries. AD is characterized pathologically by the presence of senile plaques (SPs) and neurofibrillary tangles (NFTs), the major constituents of which are amyloid β protein and tau protein, respectively. Advanced glycation end-products (AGEs), senescent macroprotein deriva...

Alternative Titles

Full title

Possible Involvement of Advanced Glycation End-Products (AGEs) in the Pathogenesis of Alzheimers Disease

Authors, Artists and Contributors

Identifiers

Primary Identifiers

Record Identifier

TN_cdi_proquest_miscellaneous_69206240

Permalink

https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_proquest_miscellaneous_69206240

Other Identifiers

ISSN

1381-6128,1873-4286

E-ISSN

1873-4286

DOI

10.2174/138161208784139693

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