Prolactin and estradiol utilize distinct mechanisms to increase serine-118 phosphorylation and decre...
Prolactin and estradiol utilize distinct mechanisms to increase serine-118 phosphorylation and decrease levels of estrogen receptor α in T47D breast cancer cells
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Publisher
Boston: Boston : Springer US
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Language
English
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Publisher
Boston: Boston : Springer US
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Contents
Potential interactions between prolactin (PRL) and estradiol (E2) in breast cancer cells were explored by examining the effect of PRL on estrogen receptor (ER) serine-118 phosphorylation, ER down-regulation, and E2-stimulated cell proliferation. Both E2 and PRL resulted in prolonged ERα serine-118 phosphorylation, but used different signaling pathw...
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Full title
Prolactin and estradiol utilize distinct mechanisms to increase serine-118 phosphorylation and decrease levels of estrogen receptor α in T47D breast cancer cells
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TN_cdi_proquest_miscellaneous_733343244
Permalink
https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_proquest_miscellaneous_733343244
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ISSN
0167-6806
E-ISSN
1573-7217
DOI
10.1007/s10549-009-0400-7
