Deficiency of the complement regulator CD59a enhances disease severity, demyelination and axonal inj...
Deficiency of the complement regulator CD59a enhances disease severity, demyelination and axonal injury in murine acute experimental allergic encephalomyelitis
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New York: Elsevier Inc
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English
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New York: Elsevier Inc
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There is a growing body of evidence implicating complement and, in particular, the terminal pathway (membrane attack complex; MAC) in inducing demyelination in multiple sclerosis and experimental allergic encephalomyelitis. In this paper, we examined the disease course and pathological changes in mice deficient in the major regulator of MAC assembl...
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Deficiency of the complement regulator CD59a enhances disease severity, demyelination and axonal injury in murine acute experimental allergic encephalomyelitis
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TN_cdi_proquest_miscellaneous_80080627
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https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_proquest_miscellaneous_80080627
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0023-6837
E-ISSN
1530-0307
DOI
10.1038/labinvest.3700015