Tyrosine Phosphorylation of the K v 2.1 Channel Contributes to Injury in Brain Ischemia
Tyrosine Phosphorylation of the K v 2.1 Channel Contributes to Injury in Brain Ischemia
About this item
Full title
Tyrosine Phosphorylation of the K v 2.1 Channel Contributes to Injury in Brain Ischemia
Author / Creator
Publisher
Switzerland
Journal title
International journal of molecular sciences, 2020-12, Vol.21 (24)
Language
English
Formats
Publication information
Publisher
Switzerland
Subjects
More information
Scope and Contents
Contents
In brain ischemia, oxidative stress induces neuronal apoptosis, which is mediated by increased activity of the voltage-gated K
channel K
2.1 and results in an efflux of intracellular K
. The molecular mechanisms underlying the regulation of K
2.1 and its activity during brain ischemia are not yet fully understood. Here this study provid...
Alternative Titles
Full title
Tyrosine Phosphorylation of the K v 2.1 Channel Contributes to Injury in Brain Ischemia
Authors, Artists and Contributors
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Primary Identifiers
Record Identifier
TN_cdi_pubmed_primary_33333928
Permalink
https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_pubmed_primary_33333928
Other Identifiers
E-ISSN
1422-0067
DOI
10.3390/ijms21249538
