Elevated SLC40A1 impairs cardiac function and exacerbates mitochondrial dysfunction, oxidative stres...
Elevated SLC40A1 impairs cardiac function and exacerbates mitochondrial dysfunction, oxidative stress, and apoptosis in ischemic myocardia
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Author / Creator
Feng, Renqian , Wang, Di , Li, Tiantian , Liu, Xulin , Peng, Tingwei , Liu, Mingchuan , Ren, Gaotong , Xu, Haowei , Luo, Haixia , Lu, Denghui , Qi, Bingchao , Zhang, Mingming and Li, Yan
Publisher
Australia: Ivyspring International Publisher
Journal title
Language
English
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Publication information
Publisher
Australia: Ivyspring International Publisher
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Scope and Contents
Contents
Iron homeostasis is crucial for optimal cardiac function. Iron deficiency and overload have been linked to the development of cardiomyopathy and heart failure (HF) via intricate mechanisms. Although the crucial role of SLC40A1 in iron metabolism by facilitating the efflux of cellular iron has been confirmed, its specific molecular functions in card...
Alternative Titles
Full title
Elevated SLC40A1 impairs cardiac function and exacerbates mitochondrial dysfunction, oxidative stress, and apoptosis in ischemic myocardia
Authors, Artists and Contributors
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Primary Identifiers
Record Identifier
TN_cdi_pubmedcentral_primary_oai_pubmedcentral_nih_gov_10758104
Permalink
https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_pubmedcentral_primary_oai_pubmedcentral_nih_gov_10758104
Other Identifiers
ISSN
1449-2288
E-ISSN
1449-2288
DOI
10.7150/ijbs.89368
