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Presynaptic mechanisms of impaired long term potentiation of lateral entorhinal cortex input to CA1...

Presynaptic mechanisms of impaired long term potentiation of lateral entorhinal cortex input to CA1...

https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_pubmedcentral_primary_oai_pubmedcentral_nih_gov_11710336

Presynaptic mechanisms of impaired long term potentiation of lateral entorhinal cortex input to CA1 in the PS19 mouse model of tauopathy

About this item

Full title

Presynaptic mechanisms of impaired long term potentiation of lateral entorhinal cortex input to CA1 in the PS19 mouse model of tauopathy

Publisher

Hoboken: John Wiley and Sons Inc

Journal title

Alzheimer's & dementia, 2024-12, Vol.20 (S1), p.n/a

Language

English

Formats

Publication information

Publisher

Hoboken: John Wiley and Sons Inc

Subjects

More information

Scope and Contents

Contents

Background
How tauopathy disrupts direct entorhinal cortex (EC) inputs to CA1 and their plasticity is understudied, despite its critical role in memory. Moreover, dysfunction of lateral EC (LEC) input is less clear, despite its relevance to early Alzheimer’s disease pathogenesis. Here we examined how tau impacts long‐term potentiation (LTP) of L...

Alternative Titles

Full title

Presynaptic mechanisms of impaired long term potentiation of lateral entorhinal cortex input to CA1 in the PS19 mouse model of tauopathy

Authors, Artists and Contributors

Identifiers

Primary Identifiers

Record Identifier

TN_cdi_pubmedcentral_primary_oai_pubmedcentral_nih_gov_11710336

Permalink

https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_pubmedcentral_primary_oai_pubmedcentral_nih_gov_11710336

Other Identifiers

ISSN

1552-5260

E-ISSN

1552-5279

DOI

10.1002/alz.086703

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