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Redundant pathways for Cdc2 activation in Xenopus oocyte: either cyclin B or Mos synthesis

Redundant pathways for Cdc2 activation in Xenopus oocyte: either cyclin B or Mos synthesis

https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_pubmedcentral_primary_oai_pubmedcentral_nih_gov_1456883

Redundant pathways for Cdc2 activation in Xenopus oocyte: either cyclin B or Mos synthesis

About this item

Full title

Redundant pathways for Cdc2 activation in Xenopus oocyte: either cyclin B or Mos synthesis

Publisher

Chichester, UK: John Wiley & Sons, Ltd

Journal title

EMBO reports, 2006-03, Vol.7 (3), p.321-325

Language

English

Formats

Publication information

Publisher

Chichester, UK: John Wiley & Sons, Ltd

More information

Scope and Contents

Contents

Xenopus
oocytes are arrested in meiotic prophase I. Progesterone induces the resumption of meiotic maturation, which requires continuous protein synthesis to bring about Cdc2 activation. The identification of the newly synthesized proteins has long been a goal. Two plausible candidates have received extensive study. The synthesis of cyclin B and...

Alternative Titles

Full title

Redundant pathways for Cdc2 activation in Xenopus oocyte: either cyclin B or Mos synthesis

Authors, Artists and Contributors

Identifiers

Primary Identifiers

Record Identifier

TN_cdi_pubmedcentral_primary_oai_pubmedcentral_nih_gov_1456883

Permalink

https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_pubmedcentral_primary_oai_pubmedcentral_nih_gov_1456883

Other Identifiers

ISSN

1469-221X

E-ISSN

1469-3178,1469-221X

DOI

10.1038/sj.embor.7400611

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