TWEAK, via its receptor Fn14, is a novel regulator of mesenchymal progenitor cells and skeletal musc...
TWEAK, via its receptor Fn14, is a novel regulator of mesenchymal progenitor cells and skeletal muscle regeneration
About this item
Full title
Author / Creator
Girgenrath, Mahasweta , Weng, Shawn , Kostek, Christine A , Browning, Beth , Wang, Monica , Brown, Sharron AN , Winkles, Jeffrey A , Michaelson, Jennifer S , Allaire, Norm , Schneider, Pascal , Scott, Martin L , Hsu, Yen-ming , Yagita, Hideo , Flavell, Richard A , Miller, Jeffrey Boone , Burkly, Linda C and Zheng, Timothy S
Publisher
Chichester, UK: John Wiley & Sons, Ltd
Journal title
Language
English
Formats
Publication information
Publisher
Chichester, UK: John Wiley & Sons, Ltd
Subjects
More information
Scope and Contents
Contents
Inflammation participates in tissue repair through multiple mechanisms including directly regulating the cell fate of resident progenitor cells critical for successful regeneration. Upon surveying target cell types of the TNF ligand TWEAK, we observed that TWEAK binds to all progenitor cells of the mesenchymal lineage and induces NF‐κB activation a...
Alternative Titles
Full title
TWEAK, via its receptor Fn14, is a novel regulator of mesenchymal progenitor cells and skeletal muscle regeneration
Authors, Artists and Contributors
Author / Creator
Identifiers
Primary Identifiers
Record Identifier
TN_cdi_pubmedcentral_primary_oai_pubmedcentral_nih_gov_1698888
Permalink
https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_pubmedcentral_primary_oai_pubmedcentral_nih_gov_1698888
Other Identifiers
ISSN
0261-4189
E-ISSN
1460-2075
DOI
10.1038/sj.emboj.7601441