ATR-dependent phosphorylation and activation of ATM in response to UV treatment or replication fork...
ATR-dependent phosphorylation and activation of ATM in response to UV treatment or replication fork stalling
About this item
Full title
Author / Creator
Publisher
Chichester, UK: John Wiley & Sons, Ltd
Journal title
Language
English
Formats
Publication information
Publisher
Chichester, UK: John Wiley & Sons, Ltd
Subjects
More information
Scope and Contents
Contents
The phosphatidyl inositol 3‐kinase‐like kinases (PIKKs), ataxia‐telangiectasia mutated (ATM) and ATM‐ and Rad3‐related (ATR) regulate parallel damage response signalling pathways. ATM is reported to be activated by DNA double‐strand breaks (DSBs), whereas ATR is recruited to single‐stranded regions of DNA. Although the two pathways were considered...
Alternative Titles
Full title
ATR-dependent phosphorylation and activation of ATM in response to UV treatment or replication fork stalling
Authors, Artists and Contributors
Identifiers
Primary Identifiers
Record Identifier
TN_cdi_pubmedcentral_primary_oai_pubmedcentral_nih_gov_1698893
Permalink
https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_pubmedcentral_primary_oai_pubmedcentral_nih_gov_1698893
Other Identifiers
ISSN
0261-4189
E-ISSN
1460-2075
DOI
10.1038/sj.emboj.7601446
