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Vascular and inflammatory stresses mediate atherosclerosis via RAGE and its ligands in apoE–/– mice

Vascular and inflammatory stresses mediate atherosclerosis via RAGE and its ligands in apoE–/– mice

https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_pubmedcentral_primary_oai_pubmedcentral_nih_gov_2129235

Vascular and inflammatory stresses mediate atherosclerosis via RAGE and its ligands in apoE–/– mice

About this item

Full title

Vascular and inflammatory stresses mediate atherosclerosis via RAGE and its ligands in apoE–/– mice

Publisher

United States: American Society for Clinical Investigation

Journal title

The Journal of clinical investigation, 2008-01, Vol.118 (1), p.183-194

Language

English

Formats

Publication information

Publisher

United States: American Society for Clinical Investigation

More information

Scope and Contents

Contents

Endothelial dysfunction is a key triggering event in atherosclerosis. Following the entry of lipoproteins into the vessel wall, their rapid modification results in the generation of advanced glycation endproduct epitopes and subsequent infiltration of inflammatory cells. These inflammatory cells release receptor for advanced glycation endproduct (R...

Alternative Titles

Full title

Vascular and inflammatory stresses mediate atherosclerosis via RAGE and its ligands in apoE–/– mice

Identifiers

Primary Identifiers

Record Identifier

TN_cdi_pubmedcentral_primary_oai_pubmedcentral_nih_gov_2129235

Permalink

https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_pubmedcentral_primary_oai_pubmedcentral_nih_gov_2129235

Other Identifiers

ISSN

0021-9738

DOI

10.1172/JCI32703

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