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Evidence of Dysfunction of Endothelial Progenitors in Pulmonary Arterial Hypertension

Evidence of Dysfunction of Endothelial Progenitors in Pulmonary Arterial Hypertension

https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_pubmedcentral_primary_oai_pubmedcentral_nih_gov_2778151

Evidence of Dysfunction of Endothelial Progenitors in Pulmonary Arterial Hypertension

About this item

Full title

Evidence of Dysfunction of Endothelial Progenitors in Pulmonary Arterial Hypertension

Publisher

New York, NY: Am Thoracic Soc

Journal title

American journal of respiratory and critical care medicine, 2009-10, Vol.180 (8), p.780-787

Language

English

Formats

Publication information

Publisher

New York, NY: Am Thoracic Soc

More information

Scope and Contents

Contents

Severe pulmonary arterial hypertension (PAH) is characterized by the formation of plexiform lesions and concentric intimal fibrosis in small pulmonary arteries. The origin of cells contributing to these vascular lesions is uncertain. Endogenous endothelial progenitor cells are potential contributors to this process.
To determine whether progenit...

Alternative Titles

Full title

Evidence of Dysfunction of Endothelial Progenitors in Pulmonary Arterial Hypertension

Identifiers

Primary Identifiers

Record Identifier

TN_cdi_pubmedcentral_primary_oai_pubmedcentral_nih_gov_2778151

Permalink

https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_pubmedcentral_primary_oai_pubmedcentral_nih_gov_2778151

Other Identifiers

ISSN

1073-449X

E-ISSN

1535-4970

DOI

10.1164/rccm.200810-1662OC

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