Neural Mechanisms of Angiotensin II–Salt Hypertension: Implications for Therapies Targeting Neural C...
Neural Mechanisms of Angiotensin II–Salt Hypertension: Implications for Therapies Targeting Neural Control of the Splanchnic Circulation
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Publisher
New York: Current Science Inc
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Language
English
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New York: Current Science Inc
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Contents
Chronically elevated plasma angiotensin II (AngII) causes a salt-sensitive form of hypertension that is associated with a differential pattern of peripheral sympathetic outflow. This “AngII-salt sympathetic signature” is characterized by a transient reduction in sympathetic nervous system activity (SNA) to the kidneys, no change in SNA to skeletal...
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Full title
Neural Mechanisms of Angiotensin II–Salt Hypertension: Implications for Therapies Targeting Neural Control of the Splanchnic Circulation
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TN_cdi_pubmedcentral_primary_oai_pubmedcentral_nih_gov_3076522
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https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_pubmedcentral_primary_oai_pubmedcentral_nih_gov_3076522
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ISSN
1522-6417
E-ISSN
1534-3111
DOI
10.1007/s11906-011-0188-9
