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Neovascularization of coronary tunica intima (DIT) is the cause of coronary atherosclerosis. Lipopro...

Neovascularization of coronary tunica intima (DIT) is the cause of coronary atherosclerosis. Lipopro...

https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_pubmedcentral_primary_oai_pubmedcentral_nih_gov_3492120

Neovascularization of coronary tunica intima (DIT) is the cause of coronary atherosclerosis. Lipoproteins invade coronary intima via neovascularization from adventitial vasa vasorum, but not from the arterial lumen: a hypothesis

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Full title

Neovascularization of coronary tunica intima (DIT) is the cause of coronary atherosclerosis. Lipoproteins invade coronary intima via neovascularization from adventitial vasa vasorum, but not from the arterial lumen: a hypothesis

Author / Creator

Publisher

England: BioMed Central Ltd

Journal title

Theoretical biology and medical modelling, 2012-04, Vol.9 (1), p.11-11, Article 11

Language

English

Formats

Publication information

Publisher

England: BioMed Central Ltd

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Scope and Contents

Contents

An accepted hypothesis states that coronary atherosclerosis (CA) is initiated by endothelial dysfunction due to inflammation and high levels of LDL-C, followed by deposition of lipids and macrophages from the luminal blood into the arterial intima, resulting in plaque formation. The success of statins in preventing CA promised much for extended pro...

Alternative Titles

Full title

Neovascularization of coronary tunica intima (DIT) is the cause of coronary atherosclerosis. Lipoproteins invade coronary intima via neovascularization from adventitial vasa vasorum, but not from the arterial lumen: a hypothesis

Authors, Artists and Contributors

Author / Creator

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Primary Identifiers

Record Identifier

TN_cdi_pubmedcentral_primary_oai_pubmedcentral_nih_gov_3492120

Permalink

https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_pubmedcentral_primary_oai_pubmedcentral_nih_gov_3492120

Other Identifiers

ISSN

1742-4682

E-ISSN

1742-4682

DOI

10.1186/1742-4682-9-11

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