Polymorphisms at the innate immune receptor TLR2 are associated with Borrelia infection in a wild ro...
Polymorphisms at the innate immune receptor TLR2 are associated with Borrelia infection in a wild rodent population
About this item
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Author / Creator
Naturvetenskapliga fakulteten , Molecular Ecology and Evolution Lab , Lund University , MEMEG , Biologiska institutionen , Forskargrupper vid Biologiska institutionen , Molekylär ekologi och evolution , Department of Biology , Research groups at the Department of Biology , Faculty of Science , Lunds universitet , Tschirren, Barbara , Andersson, Martin , Scherman, Kristin , Westerdahl, Helena , Mittl, Peer R. E. and Råberg, Lars
Publisher
England: The Royal Society
Journal title
Language
English
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Publication information
Publisher
England: The Royal Society
Subjects
More information
Scope and Contents
Contents
The discovery of the key role of Toll-like receptors (TLRs) in initiating innate immune responses and modulating adaptive immunity has revolutionized our understanding of vertebrate defence against pathogens. Yet, despite their central role in pathogen recognition and defence initiation, there is little information on how variation in TLRs influenc...
Alternative Titles
Full title
Polymorphisms at the innate immune receptor TLR2 are associated with Borrelia infection in a wild rodent population
Authors, Artists and Contributors
Author / Creator
Molecular Ecology and Evolution Lab
Lund University
MEMEG
Biologiska institutionen
Forskargrupper vid Biologiska institutionen
Molekylär ekologi och evolution
Department of Biology
Research groups at the Department of Biology
Faculty of Science
Lunds universitet
Tschirren, Barbara
Andersson, Martin
Scherman, Kristin
Westerdahl, Helena
Mittl, Peer R. E.
Råberg, Lars
Identifiers
Primary Identifiers
Record Identifier
TN_cdi_pubmedcentral_primary_oai_pubmedcentral_nih_gov_3619520
Permalink
https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_pubmedcentral_primary_oai_pubmedcentral_nih_gov_3619520
Other Identifiers
ISSN
0962-8452,1471-2954
E-ISSN
DOI
10.1098/rspb.2013.0364
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