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Microglia, Amyloid, and Glucose Metabolism in Parkinson’s Disease with and without Dementia

Microglia, Amyloid, and Glucose Metabolism in Parkinson’s Disease with and without Dementia

https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_pubmedcentral_primary_oai_pubmedcentral_nih_gov_3629382

Microglia, Amyloid, and Glucose Metabolism in Parkinson’s Disease with and without Dementia

About this item

Full title

Microglia, Amyloid, and Glucose Metabolism in Parkinson’s Disease with and without Dementia

Publisher

Basingstoke: Nature Publishing Group

Journal title

Neuropsychopharmacology (New York, N.Y.), 2013-05, Vol.38 (6), p.938-949

Language

English

Formats

Publication information

Publisher

Basingstoke: Nature Publishing Group

More information

Scope and Contents

Contents

[(11)C](R)PK11195-PET measures upregulation of translocator protein, which is associated with microglial activation, [(11)C]PIB-PET is a marker of amyloid, while [(18)F]FDG-PET measures cerebral glucose metabolism (rCMRGlc). We hypothesize that microglial activation is an early event in the Parkinson's disease (PD) spectrum and is independent of th...

Alternative Titles

Full title

Microglia, Amyloid, and Glucose Metabolism in Parkinson’s Disease with and without Dementia

Identifiers

Primary Identifiers

Record Identifier

TN_cdi_pubmedcentral_primary_oai_pubmedcentral_nih_gov_3629382

Permalink

https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_pubmedcentral_primary_oai_pubmedcentral_nih_gov_3629382

Other Identifiers

ISSN

0893-133X

E-ISSN

1740-634X

DOI

10.1038/npp.2012.255

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