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A type III effector antagonizes death receptor signalling during bacterial gut infection

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A type III effector antagonizes death receptor signalling during bacterial gut infection

https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_pubmedcentral_primary_oai_pubmedcentral_nih_gov_3836246

A type III effector antagonizes death receptor signalling during bacterial gut infection

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Publication information

Publisher

London: Nature Publishing Group UK

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Subjects and topics

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Scope and Contents

Contents

Colonizing enteric bacteria are shown to inhibit the antimicrobial process of host cell apoptosis through the action of NleB1, a type III secretion system effector with
N-
acetylglucosamine transferase activity, which can bind and modify eukaryotic death-domain-containing proteins.
Virulence mechanism of bacterial effector NleB
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Alternative Titles

Full title

A type III effector antagonizes death receptor signalling during bacterial gut infection

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Record Identifier

TN_cdi_pubmedcentral_primary_oai_pubmedcentral_nih_gov_3836246

Permalink

https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_pubmedcentral_primary_oai_pubmedcentral_nih_gov_3836246

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ISSN

0028-0836

E-ISSN

1476-4687

DOI

10.1038/nature12524

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