Depletion of circulating blood NOS3 increases severity of myocardial infarction and left ventricular...
Depletion of circulating blood NOS3 increases severity of myocardial infarction and left ventricular dysfunction
About this item
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Author / Creator
Publisher
Berlin/Heidelberg: Springer Berlin Heidelberg
Journal title
Language
English
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Publication information
Publisher
Berlin/Heidelberg: Springer Berlin Heidelberg
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More information
Scope and Contents
Contents
Nitric oxide (NO) derived from endothelial NO synthase (NOS3) plays a central role in myocardial ischemia/reperfusion (I/R)-injury. Subsets of circulating blood cells, including red blood cells (RBCs), carry a NOS3 and contribute to blood pressure regulation and RBC nitrite/nitrate formation. We hypothesized that the circulating blood born NOS3 als...
Alternative Titles
Full title
Depletion of circulating blood NOS3 increases severity of myocardial infarction and left ventricular dysfunction
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Record Identifier
TN_cdi_pubmedcentral_primary_oai_pubmedcentral_nih_gov_3898535
Permalink
https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_pubmedcentral_primary_oai_pubmedcentral_nih_gov_3898535
Other Identifiers
ISSN
0300-8428,1435-1803
E-ISSN
1435-1803
DOI
10.1007/s00395-013-0398-1
