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Mechanisms of CaMKII action in long-term potentiation

Mechanisms of CaMKII action in long-term potentiation

https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_pubmedcentral_primary_oai_pubmedcentral_nih_gov_4050655

Mechanisms of CaMKII action in long-term potentiation

About this item

Full title

Mechanisms of CaMKII action in long-term potentiation

Publisher

London: Nature Publishing Group UK

Journal title

Nature reviews. Neuroscience, 2012-03, Vol.13 (3), p.169-182

Language

English

Formats

Publication information

Publisher

London: Nature Publishing Group UK

More information

Scope and Contents

Contents

Key Points
The mechanisms that result in synapse-specific activation of calcium/calmodulin-dependent protein kinase II (CaMKII) have been determined.
CaMKII translocates to the synapse during long-term potentiation (LTP), in part owing to binding of CaMKII to the NMDA-type glutamate receptor (NMDAR).
One component of early LTP involves CaM...

Alternative Titles

Full title

Mechanisms of CaMKII action in long-term potentiation

Authors, Artists and Contributors

Identifiers

Primary Identifiers

Record Identifier

TN_cdi_pubmedcentral_primary_oai_pubmedcentral_nih_gov_4050655

Permalink

https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_pubmedcentral_primary_oai_pubmedcentral_nih_gov_4050655

Other Identifiers

ISSN

1471-003X

E-ISSN

1471-0048,1469-3178

DOI

10.1038/nrn3192

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