Claudin-18 deficiency results in alveolar barrier dysfunction and impaired alveologenesis in mice
Claudin-18 deficiency results in alveolar barrier dysfunction and impaired alveologenesis in mice
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Publisher
United States: American Thoracic Society
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Language
English
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Publisher
United States: American Thoracic Society
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Contents
Claudins are a family of transmembrane proteins that are required for tight junction formation. Claudin (CLDN)-18.1, the only known lung-specific tight junction protein, is the most abundant claudin in alveolar epithelial type (AT) 1 cells, and is regulated by lung maturational agonists and inflammatory mediators. To determine the function of CLDN1...
Alternative Titles
Full title
Claudin-18 deficiency results in alveolar barrier dysfunction and impaired alveologenesis in mice
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TN_cdi_pubmedcentral_primary_oai_pubmedcentral_nih_gov_4189483
Permalink
https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_pubmedcentral_primary_oai_pubmedcentral_nih_gov_4189483
Other Identifiers
ISSN
1044-1549
E-ISSN
1535-4989
DOI
10.1165/rcmb.2013-0456OC
