A novel form of ciliopathy underlies hyperphagia and obesity in Ankrd26 knockout mice
A novel form of ciliopathy underlies hyperphagia and obesity in Ankrd26 knockout mice
About this item
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Author / Creator
Publisher
Berlin/Heidelberg: Springer Berlin Heidelberg
Journal title
Language
English
Formats
Publication information
Publisher
Berlin/Heidelberg: Springer Berlin Heidelberg
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More information
Scope and Contents
Contents
Human ciliopathies are genetic disorders caused by mutations in genes responsible for the formation and function of primary cilia. Some are associated with hyperphagia and obesity (e.g., Bardet–Biedl Syndrome, Alström Syndrome), but the mechanisms underlying these problems are not fully understood. The human gene
ANKRD26
is located on 10p12,...
Alternative Titles
Full title
A novel form of ciliopathy underlies hyperphagia and obesity in Ankrd26 knockout mice
Authors, Artists and Contributors
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Record Identifier
TN_cdi_pubmedcentral_primary_oai_pubmedcentral_nih_gov_4601608
Permalink
https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_pubmedcentral_primary_oai_pubmedcentral_nih_gov_4601608
Other Identifiers
ISSN
1863-2653
E-ISSN
1863-2661,0340-2061
DOI
10.1007/s00429-014-0741-9
