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Increased S100A4 expression in the vasculature of human COPD lungs and murine model of smoke-induced...

Increased S100A4 expression in the vasculature of human COPD lungs and murine model of smoke-induced...

https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_pubmedcentral_primary_oai_pubmedcentral_nih_gov_4612429

Increased S100A4 expression in the vasculature of human COPD lungs and murine model of smoke-induced emphysema

About this item

Full title

Increased S100A4 expression in the vasculature of human COPD lungs and murine model of smoke-induced emphysema

Publisher

England: BioMed Central Ltd

Journal title

Respiratory research, 2015-10, Vol.16 (1), p.127-127, Article 127

Language

English

Formats

Publication information

Publisher

England: BioMed Central Ltd

More information

Scope and Contents

Contents

Chronic obstructive lung disease (COPD) is a common cause of death in industrialized countries often induced by exposure to tobacco smoke. A substantial number of patients with COPD also suffer from pulmonary hypertension that may be caused by hypoxia or other hypoxia-independent stimuli - inducing pulmonary vascular remodeling. The Ca(2+) binding...

Alternative Titles

Full title

Increased S100A4 expression in the vasculature of human COPD lungs and murine model of smoke-induced emphysema

Identifiers

Primary Identifiers

Record Identifier

TN_cdi_pubmedcentral_primary_oai_pubmedcentral_nih_gov_4612429

Permalink

https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_pubmedcentral_primary_oai_pubmedcentral_nih_gov_4612429

Other Identifiers

ISSN

1465-993X,1465-9921

E-ISSN

1465-993X,1465-9921

DOI

10.1186/s12931-015-0284-5

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