Inactivation of ID4 promotes a CRPC phenotype with constitutive AR activation through FKBP52
Inactivation of ID4 promotes a CRPC phenotype with constitutive AR activation through FKBP52
About this item
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Author / Creator
Publisher
United States: John Wiley & Sons, Inc
Journal title
Language
English
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Publisher
United States: John Wiley & Sons, Inc
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Scope and Contents
Contents
Castration‐resistant prostate cancer (CRPC) is the emergence of prostate cancer cells that have adapted to the androgen‐depleted environment of the prostate. In recent years, targeting multiple chaperones and co‐chaperones (e.g., Hsp27, FKBP52) that promote androgen receptor (AR) signaling and/or novel AR regulatory mechanisms have emerged as promi...
Alternative Titles
Full title
Inactivation of ID4 promotes a CRPC phenotype with constitutive AR activation through FKBP52
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TN_cdi_pubmedcentral_primary_oai_pubmedcentral_nih_gov_5378613
Permalink
https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_pubmedcentral_primary_oai_pubmedcentral_nih_gov_5378613
Other Identifiers
ISSN
1574-7891
E-ISSN
1878-0261
DOI
10.1002/1878-0261.12028
