BBC3 in macrophages promoted pulmonary fibrosis development through inducing autophagy during silico...
BBC3 in macrophages promoted pulmonary fibrosis development through inducing autophagy during silicosis
About this item
Full title
Author / Creator
Liu, Haijun , Cheng, Yusi , Yang, Jian , Wang, Wei , Fang, Shencun , Zhang, Wei , Han, Bing , Zhou, Zewei , Yao, Honghong , Chao, Jie and Liao, Hong
Publisher
London: Nature Publishing Group UK
Journal title
Language
English
Formats
Publication information
Publisher
London: Nature Publishing Group UK
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Scope and Contents
Contents
Following inhalation into the lungs, silica particles are engulfed by alveolar macrophages, which triggers endogenous or exogenous apoptosis signaling pathways. As an inducer of apoptosis, the role of BBC3/PUMA (BCL2-binding component 3) in macrophages during silicosis remains unknown. Here, we exposed U937 cell-derived macrophages (UDMs) to SiO
Alternative Titles
Full title
BBC3 in macrophages promoted pulmonary fibrosis development through inducing autophagy during silicosis
Authors, Artists and Contributors
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Primary Identifiers
Record Identifier
TN_cdi_pubmedcentral_primary_oai_pubmedcentral_nih_gov_5386570
Permalink
https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_pubmedcentral_primary_oai_pubmedcentral_nih_gov_5386570
Other Identifiers
ISSN
2041-4889
E-ISSN
2041-4889
DOI
10.1038/cddis.2017.78