Sodium channel NaV1.9 mutations associated with insensitivity to pain dampen neuronal excitability
Sodium channel NaV1.9 mutations associated with insensitivity to pain dampen neuronal excitability
About this item
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Publisher
United States: American Society for Clinical Investigation
Journal title
Language
English
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Publisher
United States: American Society for Clinical Investigation
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Scope and Contents
Contents
Voltage-gated sodium channel (NaV) mutations cause genetic pain disorders that range from severe paroxysmal pain to a congenital inability to sense pain. Previous studies on NaV1.7 and NaV1.8 established clear relationships between perturbations in channel function and divergent clinical phenotypes. By contrast, studies of NaV1.9 mutations have not...
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Full title
Sodium channel NaV1.9 mutations associated with insensitivity to pain dampen neuronal excitability
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TN_cdi_pubmedcentral_primary_oai_pubmedcentral_nih_gov_5490760
Permalink
https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_pubmedcentral_primary_oai_pubmedcentral_nih_gov_5490760
Other Identifiers
ISSN
0021-9738
E-ISSN
1558-8238
DOI
10.1172/JCI92373
