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Overexpression of SMC4 activates TGFβ/Smad signaling and promotes aggressive phenotype in glioma cel...

Overexpression of SMC4 activates TGFβ/Smad signaling and promotes aggressive phenotype in glioma cel...

https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_pubmedcentral_primary_oai_pubmedcentral_nih_gov_5533949

Overexpression of SMC4 activates TGFβ/Smad signaling and promotes aggressive phenotype in glioma cells

About this item

Full title

Overexpression of SMC4 activates TGFβ/Smad signaling and promotes aggressive phenotype in glioma cells

Publisher

London: Nature Publishing Group UK

Journal title

Oncogenesis (New York, NY), 2017-03, Vol.6 (3), p.e301-e301

Language

English

Formats

Publication information

Publisher

London: Nature Publishing Group UK

More information

Scope and Contents

Contents

Overexpression of structural maintenance of chromosomes 4 (SMC4) has been reported to be involved in tumor cell growth, migration and invasion, and to be correlated with poor prognosis of cancer patient. However, its clinical significance and biological role in glioma remain unknown. Herein, we found that SMC4 expression at both mRNA and protein le...

Alternative Titles

Full title

Overexpression of SMC4 activates TGFβ/Smad signaling and promotes aggressive phenotype in glioma cells

Identifiers

Primary Identifiers

Record Identifier

TN_cdi_pubmedcentral_primary_oai_pubmedcentral_nih_gov_5533949

Permalink

https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_pubmedcentral_primary_oai_pubmedcentral_nih_gov_5533949

Other Identifiers

ISSN

2157-9024

E-ISSN

2157-9024

DOI

10.1038/oncsis.2017.8

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