BAP1 regulates IP3R3-mediated Ca2+ flux to mitochondria suppressing cell transformation
BAP1 regulates IP3R3-mediated Ca2+ flux to mitochondria suppressing cell transformation
About this item
Full title
Author / Creator
Bononi, Angela , Giorgi, Carlotta , Patergnani, Simone , Larson, David , Verbruggen, Kaitlyn , Tanji, Mika , Pellegrini, Laura , Signorato, Valentina , Olivetto, Federica , Pastorino, Sandra , Nasu, Masaki , Napolitano, Andrea , Gaudino, Giovanni , Morris, Paul , Sakamoto, Greg , Ferris, Laura K. , Danese, Alberto , Raimondi, Andrea , Tacchetti, Carlo , Kuchay, Shafi , Pass, Harvey I. , Affar, El Bachir , Yang, Haining , Pinton, Paolo and Carbone, Michele
Publisher
London: Nature Publishing Group UK
Journal title
Language
English
Formats
Publication information
Publisher
London: Nature Publishing Group UK
Subjects
More information
Scope and Contents
Contents
BRCA1-associated protein 1 (BAP1) regulates calcium flux in the endoplasmic reticulum to facilitate the execution of apoptosis, unveiling a new facet of the role of BAP1 as an environmental tumour suppressor.
A new role for BAP1 in tumour suppression
BAP1 is a tumour suppressor associated with germline mutations in several malignancies includ...
Alternative Titles
Full title
BAP1 regulates IP3R3-mediated Ca2+ flux to mitochondria suppressing cell transformation
Authors, Artists and Contributors
Author / Creator
Giorgi, Carlotta
Patergnani, Simone
Larson, David
Verbruggen, Kaitlyn
Tanji, Mika
Pellegrini, Laura
Signorato, Valentina
Olivetto, Federica
Pastorino, Sandra
Nasu, Masaki
Napolitano, Andrea
Gaudino, Giovanni
Morris, Paul
Sakamoto, Greg
Ferris, Laura K.
Danese, Alberto
Raimondi, Andrea
Tacchetti, Carlo
Kuchay, Shafi
Pass, Harvey I.
Affar, El Bachir
Yang, Haining
Pinton, Paolo
Carbone, Michele
Identifiers
Primary Identifiers
Record Identifier
TN_cdi_pubmedcentral_primary_oai_pubmedcentral_nih_gov_5581194
Permalink
https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_pubmedcentral_primary_oai_pubmedcentral_nih_gov_5581194
Other Identifiers
ISSN
0028-0836
E-ISSN
1476-4687
DOI
10.1038/nature22798
