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Trichostatin A attenuates oxidative stress-mediated myocardial injury through the FoxO3a signaling p...

Trichostatin A attenuates oxidative stress-mediated myocardial injury through the FoxO3a signaling p...

https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_pubmedcentral_primary_oai_pubmedcentral_nih_gov_5593460

Trichostatin A attenuates oxidative stress-mediated myocardial injury through the FoxO3a signaling pathway

About this item

Full title

Trichostatin A attenuates oxidative stress-mediated myocardial injury through the FoxO3a signaling pathway

Publisher

Athens: Spandidos Publications UK Ltd

Journal title

International journal of molecular medicine, 2017-10, Vol.40 (4), p.999-1008

Language

English

Formats

Publication information

Publisher

Athens: Spandidos Publications UK Ltd

More information

Scope and Contents

Contents

Trichostatin A (TSA), a histone deacetylase inhibitor, is widely used as an anticancer drug. Recently, TSA has been shown to exert a protective effect on ischemia/reperfusion (I/R) injury; however, the underlying mechanisms remain unclear. Forkhead box O3a (FoxO3a), a unique FoxO family member, has been shown to attenuate myocardial injury by incre...

Alternative Titles

Full title

Trichostatin A attenuates oxidative stress-mediated myocardial injury through the FoxO3a signaling pathway

Authors, Artists and Contributors

Identifiers

Primary Identifiers

Record Identifier

TN_cdi_pubmedcentral_primary_oai_pubmedcentral_nih_gov_5593460

Permalink

https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_pubmedcentral_primary_oai_pubmedcentral_nih_gov_5593460

Other Identifiers

ISSN

1107-3756

E-ISSN

1791-244X

DOI

10.3892/ijmm.2017.3101

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