Phenytoin inhibits necroptosis
Phenytoin inhibits necroptosis
About this item
Full title
Author / Creator
von Mässenhausen, Anne , Tonnus, Wulf , Himmerkus, Nina , Parmentier, Simon , Saleh, Danish , Rodriguez, Diego , Ousingsawat, Jiraporn , Ang, Rosalind L. , Weinberg, Joel M. , Sanz, Ana B. , Ortiz, Alberto , Zierleyn, Adrian , Becker, Jan Ulrich , Baratte, Blandine , Desban, Nathalie , Bach, Stéphane , Schiessl, Ina Maria , Nogusa, Shoko , Balachandran, Siddharth , Anders, Hans Joachim , Ting, Adrian T. , Bleich, Markus , Degterev, Alexei , Kunzelmann, Karl , Bornstein, Stefan R. , Green, Douglas R. , Hugo, Christian and Linkermann, Andreas
Publisher
London: Nature Publishing Group UK
Journal title
Language
English
Formats
Publication information
Publisher
London: Nature Publishing Group UK
Subjects
More information
Scope and Contents
Contents
Receptor-interacting protein kinases 1 and 3 (RIPK1/3) have best been described for their role in mediating a regulated form of necrosis, referred to as necroptosis. During this process, RIPK3 phosphorylates mixed lineage kinase domain-like (MLKL) to cause plasma membrane rupture. RIPK3-deficient mice have recently been demonstrated to be protected...
Alternative Titles
Full title
Phenytoin inhibits necroptosis
Authors, Artists and Contributors
Author / Creator
Tonnus, Wulf
Himmerkus, Nina
Parmentier, Simon
Saleh, Danish
Rodriguez, Diego
Ousingsawat, Jiraporn
Ang, Rosalind L.
Weinberg, Joel M.
Sanz, Ana B.
Ortiz, Alberto
Zierleyn, Adrian
Becker, Jan Ulrich
Baratte, Blandine
Desban, Nathalie
Bach, Stéphane
Schiessl, Ina Maria
Nogusa, Shoko
Balachandran, Siddharth
Anders, Hans Joachim
Ting, Adrian T.
Bleich, Markus
Degterev, Alexei
Kunzelmann, Karl
Bornstein, Stefan R.
Green, Douglas R.
Hugo, Christian
Linkermann, Andreas
Identifiers
Primary Identifiers
Record Identifier
TN_cdi_pubmedcentral_primary_oai_pubmedcentral_nih_gov_5834524
Permalink
https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_pubmedcentral_primary_oai_pubmedcentral_nih_gov_5834524
Other Identifiers
ISSN
2041-4889
E-ISSN
2041-4889
DOI
10.1038/s41419-018-0394-3
