BP180 dysfunction triggers spontaneous skin inflammation in mice
BP180 dysfunction triggers spontaneous skin inflammation in mice
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Publisher
United States: National Academy of Sciences
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Language
English
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United States: National Academy of Sciences
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Contents
BP180, also known as collagen XVII, is a hemidesmosomal component and plays a key role in maintaining skin dermal/epidermal adhesion. Dysfunction of BP180, either through genetic mutations in junctional epidermolysis bullosa (JEB) or autoantibody insult in bullous pemphigoid (BP), leads to subepidermal blistering accompanied by skin inflammation. H...
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Full title
BP180 dysfunction triggers spontaneous skin inflammation in mice
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Record Identifier
TN_cdi_pubmedcentral_primary_oai_pubmedcentral_nih_gov_6016813
Permalink
https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_pubmedcentral_primary_oai_pubmedcentral_nih_gov_6016813
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ISSN
0027-8424
E-ISSN
1091-6490
DOI
10.1073/pnas.1721805115
