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Osteopontin isoforms differentially promote arteriogenesis in response to ischemia via macrophage ac...

Osteopontin isoforms differentially promote arteriogenesis in response to ischemia via macrophage ac...

https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_pubmedcentral_primary_oai_pubmedcentral_nih_gov_6311150

Osteopontin isoforms differentially promote arteriogenesis in response to ischemia via macrophage accumulation and survival

About this item

Full title

Osteopontin isoforms differentially promote arteriogenesis in response to ischemia via macrophage accumulation and survival

Publisher

New York: Elsevier Inc

Journal title

Laboratory investigation, 2019-03, Vol.99 (3), p.331-345

Language

English

Formats

Publication information

Publisher

New York: Elsevier Inc

More information

Scope and Contents

Contents

Osteopontin (OPN) is critical for ischemia-induced neovascularization. Unlike rodents, humans express three OPN isoforms (a, b, and c); however, the roles of these isoforms in post-ischemic neovascularization and cell migration remain undefined. Our objective was to determine if OPN isoforms differentially affect post-ischemic neovascularization an...

Alternative Titles

Full title

Osteopontin isoforms differentially promote arteriogenesis in response to ischemia via macrophage accumulation and survival

Identifiers

Primary Identifiers

Record Identifier

TN_cdi_pubmedcentral_primary_oai_pubmedcentral_nih_gov_6311150

Permalink

https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_pubmedcentral_primary_oai_pubmedcentral_nih_gov_6311150

Other Identifiers

ISSN

0023-6837

E-ISSN

1530-0307

DOI

10.1038/s41374-018-0094-8

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