Lack of IL-17 Receptor A signaling aggravates lymphoproliferation in C57BL/6 lpr mice
Lack of IL-17 Receptor A signaling aggravates lymphoproliferation in C57BL/6 lpr mice
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London: Nature Publishing Group UK
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English
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London: Nature Publishing Group UK
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Defects in Fas function correlate with susceptibility to systemic autoimmune diseases like autoimmune lymphoproliferative syndrome (ALPS) and systemic lupus erythematosus (SLE). C57BL/6 lpr (B6/lpr) mice are used as an animal model of ALPS and develop a mild SLE phenotype. Involvement of interleukin-17A (IL-17A) has been suggested in both phenotype...
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Lack of IL-17 Receptor A signaling aggravates lymphoproliferation in C57BL/6 lpr mice
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TN_cdi_pubmedcentral_primary_oai_pubmedcentral_nih_gov_6412096
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https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_pubmedcentral_primary_oai_pubmedcentral_nih_gov_6412096
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ISSN
2045-2322
E-ISSN
2045-2322
DOI
10.1038/s41598-019-39483-w
