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Intracellular Heat Shock Protein 70 Deficiency in Pulmonary Fibrosis

Intracellular Heat Shock Protein 70 Deficiency in Pulmonary Fibrosis

https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_pubmedcentral_primary_oai_pubmedcentral_nih_gov_6543745

Intracellular Heat Shock Protein 70 Deficiency in Pulmonary Fibrosis

About this item

Full title

Intracellular Heat Shock Protein 70 Deficiency in Pulmonary Fibrosis

Publisher

United States: American Thoracic Society

Journal title

American journal of respiratory cell and molecular biology, 2019-06, Vol.60 (6), p.629-636

Language

English

Formats

Publication information

Publisher

United States: American Thoracic Society

More information

Scope and Contents

Contents

Idiopathic pulmonary fibrosis (IPF) pathogenesis has been postulated to involve a variety of mechanisms associated with the aging process, including loss of protein homeostasis (proteostasis). Heat shock proteins are cellular chaperones that serve a number of vital maintenance and repair functions, including the regulation of proteostasis. Previous...

Alternative Titles

Full title

Intracellular Heat Shock Protein 70 Deficiency in Pulmonary Fibrosis

Identifiers

Primary Identifiers

Record Identifier

TN_cdi_pubmedcentral_primary_oai_pubmedcentral_nih_gov_6543745

Permalink

https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_pubmedcentral_primary_oai_pubmedcentral_nih_gov_6543745

Other Identifiers

ISSN

1044-1549

E-ISSN

1535-4989

DOI

10.1165/rcmb.2017-0268OC

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