The role of the LncRNA-FA2H-2-MLKL pathway in atherosclerosis by regulation of autophagy flux and in...
The role of the LncRNA-FA2H-2-MLKL pathway in atherosclerosis by regulation of autophagy flux and inflammation through mTOR-dependent signaling
About this item
Full title
Author / Creator
Guo, Feng-Xia , Wu, Qian , Li, Pan , Zheng, Lei , Ye, Shu , Dai, Xiao-Yan , Kang, Chun-Min , Lu, Jing-Bo , Xu, Bang-Ming , Xu, Yuan-Jun , Xiao, Lei , Lu, Zhi-Feng , Bai, Huan-Lan , Hu, Yan-Wei and Wang, Qian
Publisher
London: Nature Publishing Group UK
Journal title
Language
English
Formats
Publication information
Publisher
London: Nature Publishing Group UK
Subjects
More information
Scope and Contents
Contents
Atherosclerosis is a progressive, chronic inflammation in arterial walls. Long noncoding RNAs (lncRNAs) participate in inflammation, but the exact mechanism in atherosclerosis is unclear. Our microarray analyses revealed that the levels of lncRNA-FA2H-2 were significantly decreased by oxidized low-density lipoprotein (OX-LDL). Bioinformatics analys...
Alternative Titles
Full title
The role of the LncRNA-FA2H-2-MLKL pathway in atherosclerosis by regulation of autophagy flux and inflammation through mTOR-dependent signaling
Authors, Artists and Contributors
Identifiers
Primary Identifiers
Record Identifier
TN_cdi_pubmedcentral_primary_oai_pubmedcentral_nih_gov_6748100
Permalink
https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_pubmedcentral_primary_oai_pubmedcentral_nih_gov_6748100
Other Identifiers
ISSN
1350-9047,1476-5403
E-ISSN
1476-5403
DOI
10.1038/s41418-018-0235-z
