N-Octanoyl-Dopamine inhibits cytokine production in activated T-cells and diminishes MHC-class-II ex...
N-Octanoyl-Dopamine inhibits cytokine production in activated T-cells and diminishes MHC-class-II expression as well as adhesion molecules in IFNγ-stimulated endothelial cells
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London: Nature Publishing Group UK
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English
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London: Nature Publishing Group UK
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IFNγ enhances allograft immunogenicity and facilitates T-cell mediated rejection. This may cause interstitial fibrosis and tubular atrophy (IFTA), contributing to chronic allograft loss. We assessed if inhibition of T-cell activation by N-octanoyl dopamine (NOD) impairs adherence of activated T-cells to endothelial cells and the ability of activate...
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Full title
N-Octanoyl-Dopamine inhibits cytokine production in activated T-cells and diminishes MHC-class-II expression as well as adhesion molecules in IFNγ-stimulated endothelial cells
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TN_cdi_pubmedcentral_primary_oai_pubmedcentral_nih_gov_6920350
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https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_pubmedcentral_primary_oai_pubmedcentral_nih_gov_6920350
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ISSN
2045-2322
E-ISSN
2045-2322
DOI
10.1038/s41598-019-55983-1
