Continuous endoglin (CD105) overexpression disrupts angiogenesis and facilitates tumor cell metastas...
Continuous endoglin (CD105) overexpression disrupts angiogenesis and facilitates tumor cell metastasis
About this item
Full title
Author / Creator
Publisher
Dordrecht: Springer Netherlands
Journal title
Language
English
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Publisher
Dordrecht: Springer Netherlands
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Scope and Contents
Contents
Endoglin (CD105) is an auxiliary receptor for members of the TFG-β superfamily. Whereas it has been demonstrated that the deficiency of endoglin leads to minor and defective angiogenesis, little is known about the effect of its increased expression, characteristic of several types of cancer. Angiogenesis is essential for tumor growth, so high level...
Alternative Titles
Full title
Continuous endoglin (CD105) overexpression disrupts angiogenesis and facilitates tumor cell metastasis
Authors, Artists and Contributors
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Record Identifier
TN_cdi_pubmedcentral_primary_oai_pubmedcentral_nih_gov_7160077
Permalink
https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_pubmedcentral_primary_oai_pubmedcentral_nih_gov_7160077
Other Identifiers
ISSN
0969-6970
E-ISSN
1573-7209
DOI
10.1007/s10456-019-09703-y
