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Spironolactone Ameliorates Senescence and Calcification by Modulating Autophagy in Rat Tendon-Derive...

Spironolactone Ameliorates Senescence and Calcification by Modulating Autophagy in Rat Tendon-Derive...

https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_pubmedcentral_primary_oai_pubmedcentral_nih_gov_8263237

Spironolactone Ameliorates Senescence and Calcification by Modulating Autophagy in Rat Tendon-Derived Stem Cells via the NF-κB/MAPK Pathway

About this item

Full title

Spironolactone Ameliorates Senescence and Calcification by Modulating Autophagy in Rat Tendon-Derived Stem Cells via the NF-κB/MAPK Pathway

Publisher

United States: Hindawi

Journal title

Oxidative medicine and cellular longevity, 2021, Vol.2021 (1), p.5519587

Language

English

Formats

Publication information

Publisher

United States: Hindawi

More information

Scope and Contents

Contents

Tendinopathy is a disabling musculoskeletal disease, the pathological process of which is tightly associated with inflammation. Spironolactone (SP) has been widely used as a diuretic in clinical practice. Recently, SP has shown anti-inflammatory features in several diseases. Tendon-derived stem cells (TDSCs), a subset cell type from tendon tissue p...

Alternative Titles

Full title

Spironolactone Ameliorates Senescence and Calcification by Modulating Autophagy in Rat Tendon-Derived Stem Cells via the NF-κB/MAPK Pathway

Identifiers

Primary Identifiers

Record Identifier

TN_cdi_pubmedcentral_primary_oai_pubmedcentral_nih_gov_8263237

Permalink

https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_pubmedcentral_primary_oai_pubmedcentral_nih_gov_8263237

Other Identifiers

ISSN

1942-0900,1942-0994

E-ISSN

1942-0994

DOI

10.1155/2021/5519587

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