PERK is a critical metabolic hub for immunosuppressive function in macrophages
PERK is a critical metabolic hub for immunosuppressive function in macrophages
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Publisher
New York: Nature Publishing Group US
Journal title
Language
English
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Publisher
New York: Nature Publishing Group US
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Scope and Contents
Contents
Chronic inflammation triggers compensatory immunosuppression to stop inflammation and minimize tissue damage. Studies have demonstrated that endoplasmic reticulum (ER) stress augments the suppressive phenotypes of immune cells; however, the molecular mechanisms underpinning this process and how it links to the metabolic reprogramming of immunosuppr...
Alternative Titles
Full title
PERK is a critical metabolic hub for immunosuppressive function in macrophages
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TN_cdi_pubmedcentral_primary_oai_pubmedcentral_nih_gov_9112288
Permalink
https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_pubmedcentral_primary_oai_pubmedcentral_nih_gov_9112288
Other Identifiers
ISSN
1529-2908
E-ISSN
1529-2916
DOI
10.1038/s41590-022-01145-x
