The structure-specific endonuclease complex SLX4–XPF regulates Tus–Ter-induced homologous recombinat...
The structure-specific endonuclease complex SLX4–XPF regulates Tus–Ter-induced homologous recombination
About this item
Full title
Author / Creator
Publisher
New York: Nature Publishing Group US
Journal title
Language
English
Formats
Publication information
Publisher
New York: Nature Publishing Group US
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Scope and Contents
Contents
Vertebrate replication forks arrested at interstrand DNA cross-links (ICLs) engage the Fanconi anemia pathway to incise arrested forks, ‘unhooking’ the ICL and forming a double strand break (DSB) that is repaired by homologous recombination (HR). The
FANCP
product, SLX4, in complex with the XPF (also known as FANCQ or ERCC4)–ERCC1 endonucleas...
Alternative Titles
Full title
The structure-specific endonuclease complex SLX4–XPF regulates Tus–Ter-induced homologous recombination
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Record Identifier
TN_cdi_pubmedcentral_primary_oai_pubmedcentral_nih_gov_9941964
Permalink
https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_pubmedcentral_primary_oai_pubmedcentral_nih_gov_9941964
Other Identifiers
ISSN
1545-9993
E-ISSN
1545-9985
DOI
10.1038/s41594-022-00812-9
