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Targeting Complement C3a Receptor to Improve Outcome After Ischemic Brain Injury

Targeting Complement C3a Receptor to Improve Outcome After Ischemic Brain Injury

https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_swepub_primary_oai_gup_ub_gu_se_309131

Targeting Complement C3a Receptor to Improve Outcome After Ischemic Brain Injury

About this item

Full title

Targeting Complement C3a Receptor to Improve Outcome After Ischemic Brain Injury

Publisher

New York: Springer US

Journal title

Neurochemical research, 2021-10, Vol.46 (10), p.2626-2637

Language

English

Formats

Publication information

Publisher

New York: Springer US

More information

Scope and Contents

Contents

Ischemic stroke is a major cause of disability. No efficient therapy is currently available, except for the removal of the occluding blood clot during the first hours after symptom onset. Loss of function after stroke is due to cell death in the infarcted tissue, cell dysfunction in the peri-infarct region, as well as dysfunction and neurodegenerat...

Alternative Titles

Full title

Targeting Complement C3a Receptor to Improve Outcome After Ischemic Brain Injury

Authors, Artists and Contributors

Identifiers

Primary Identifiers

Record Identifier

TN_cdi_swepub_primary_oai_gup_ub_gu_se_309131

Permalink

https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_swepub_primary_oai_gup_ub_gu_se_309131

Other Identifiers

ISSN

0364-3190,1573-6903

E-ISSN

1573-6903

DOI

10.1007/s11064-021-03419-6

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