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ZMAT3 hypomethylation contributes to early senescence of preadipocytes from healthy first‐degree rel...

ZMAT3 hypomethylation contributes to early senescence of preadipocytes from healthy first‐degree rel...

https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_swepub_primary_oai_gup_ub_gu_se_314219

ZMAT3 hypomethylation contributes to early senescence of preadipocytes from healthy first‐degree relatives of type 2 diabetics

About this item

Full title

ZMAT3 hypomethylation contributes to early senescence of preadipocytes from healthy first‐degree relatives of type 2 diabetics

Publisher

England: John Wiley & Sons, Inc

Journal title

Aging cell, 2022-03, Vol.21 (3), p.e13557-n/a

Language

English

Formats

Publication information

Publisher

England: John Wiley & Sons, Inc

More information

Scope and Contents

Contents

Senescence of adipose precursor cells (APC) impairs adipogenesis, contributes to the age‐related subcutaneous adipose tissue (SAT) dysfunction, and increases risk of type 2 diabetes (T2D). First‐degree relatives of T2D individuals (FDR) feature restricted adipogenesis, reflecting the detrimental effects of APC senescence earlier in life and renderi...

Alternative Titles

Full title

ZMAT3 hypomethylation contributes to early senescence of preadipocytes from healthy first‐degree relatives of type 2 diabetics

Identifiers

Primary Identifiers

Record Identifier

TN_cdi_swepub_primary_oai_gup_ub_gu_se_314219

Permalink

https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_swepub_primary_oai_gup_ub_gu_se_314219

Other Identifiers

ISSN

1474-9718,1474-9726

E-ISSN

1474-9726

DOI

10.1111/acel.13557

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