Early postnatal behavioral, cellular, and molecular changes in models of Huntington disease are reve...
Early postnatal behavioral, cellular, and molecular changes in models of Huntington disease are reversible by HDAC inhibition
About this item
Full title
Author / Creator
Siebzehnrübl, Florian A. , Raber, Kerstin A. , Urbach, Yvonne K. , Schulze-Krebs, Anja , Canneva, Fabio , Moceri, Sandra , Habermeyer, Johanna , Achoui, Dalila , Gupta, Bhavana , Steindler, Dennis A. , Stephan, Michael , Nguyen, Huu Phuc , Bonin, Michael , Riess, Olaf , Bauer, Andreas , Aigner, Ludwig , Couillard-Despres, Sebastien , Paucar, Martin Arce , Svenningsson, Per , Osmand, Alexander , Andreew, Alexander , Zabel, Claus , Weiss, Andreas , Kuhn, Rainer , Moussaoui, Saliha , Blockx, Ines , Van der Linden, Annemie , Cheong, Rachel Y. , Roybon, Laurent , Petersén, Åsa , von Hörsten, Stephan , Faculty of Medicine , Medicinska fakulteten , Strategiska forskningsområden (SFO) , Department of Experimental Medical Science , Strategic research areas (SRA) , Translational Neuroendocrinology , Lunds universitet , Profilområden och andra starka forskningsmiljöer , Lund University , MultiPark: Multidisciplinary research focused on Parkinson´s disease , Institutionen för experimentell medicinsk vetenskap , Profile areas and other strong research environments , Translationell neuroendokrinologi , IPSC Laboratory for CNS Disease Modeling and Stamcellslaboratoriet för sjukdomsmodellering i det centrala nervsystemet
Publisher
United States: National Academy of Sciences
Journal title
Language
English
Formats
Publication information
Publisher
United States: National Academy of Sciences
Subjects
More information
Scope and Contents
Contents
Huntington disease (HD) is an autosomal dominant neurodegenerative disorder caused by expanded CAG repeats in the huntingtin gene (HTT). Although mutant HTT is expressed during embryonic development and throughout life, clinical HD usually manifests later in adulthood. A number of studies document neurodevelopmental changes associated with mutant H...
Alternative Titles
Full title
Early postnatal behavioral, cellular, and molecular changes in models of Huntington disease are reversible by HDAC inhibition
Authors, Artists and Contributors
Author / Creator
Raber, Kerstin A.
Urbach, Yvonne K.
Schulze-Krebs, Anja
Canneva, Fabio
Moceri, Sandra
Habermeyer, Johanna
Achoui, Dalila
Gupta, Bhavana
Steindler, Dennis A.
Stephan, Michael
Nguyen, Huu Phuc
Bonin, Michael
Riess, Olaf
Bauer, Andreas
Aigner, Ludwig
Couillard-Despres, Sebastien
Paucar, Martin Arce
Svenningsson, Per
Osmand, Alexander
Andreew, Alexander
Zabel, Claus
Weiss, Andreas
Kuhn, Rainer
Moussaoui, Saliha
Blockx, Ines
Van der Linden, Annemie
Cheong, Rachel Y.
Roybon, Laurent
Petersén, Åsa
von Hörsten, Stephan
Faculty of Medicine
Medicinska fakulteten
Strategiska forskningsområden (SFO)
Department of Experimental Medical Science
Strategic research areas (SRA)
Translational Neuroendocrinology
Lunds universitet
Profilområden och andra starka forskningsmiljöer
Lund University
MultiPark: Multidisciplinary research focused on Parkinson´s disease
Institutionen för experimentell medicinsk vetenskap
Profile areas and other strong research environments
Translationell neuroendokrinologi
IPSC Laboratory for CNS Disease Modeling
Stamcellslaboratoriet för sjukdomsmodellering i det centrala nervsystemet
Identifiers
Primary Identifiers
Record Identifier
TN_cdi_swepub_primary_oai_swepub_ki_se_485982
Permalink
https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_swepub_primary_oai_swepub_ki_se_485982
Other Identifiers
ISSN
0027-8424
E-ISSN
1091-6490
DOI
10.1073/pnas.1807962115
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