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ArticlesPeer ReviewedFull Text
Epithelial cells remove precancerous cells by cell competition via MHC class I–LILRB3 interaction
Author / Creator:Ayukawa, Shiyu ; Kamoshita, Nagisa ; Nakayama, Jun ; Teramoto, Ryohei ; Pishesha, Novalia ; Ohba, Kenji ; Sato, Nanami ; Kozawa, Kei ; Abe, Hikari ; Semba, Kentaro ; Goda, Nobuhito ; Fujita, Yasuyuki ; Maruyama, Takeshi
Publisher:New York: Nature Publishing Group US
Journal title:Nature immunology, 2021-11, Vol.22 (11), p.1391-1402
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Engineered red blood cells as carriers for systemic delivery of a wide array of functional probes
Author / Creator:Shi, Jiahai ; Kundrat, Lenka ; Pishesh, Novalia ; Bilate, Angelina ; Theile, Chris ; Maruyama, Takeshi ; Dougan, Stephanie K. ; Ploegh, Hidde L. ; Lodish, Harvey F.
Publisher:United States: National Academy of Sciences
Journal title:Proceedings of the National Academy of Sciences - PNAS, 2014-07, Vol.111 (28), p.10131-10136
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Engineered erythrocytes covalently linked to antigenic peptides can protect against autoimmune disease
Author / Creator:Pishesha, Novalia ; Bilate, Angelina M. ; Wibowo, Marsha C. ; Huang, Nai-Jia ; Li, Zeyang ; Deshycka, Rhogerry ; Bousbaine, Djenet ; Li, Hojun ; Patterson, Heide C. ; Dougan, Stephanie K. ; Maruyama, Takeshi ; Lodish, Harvey F. ; Ploegh, Hidde L.
Publisher:United States: National Academy of Sciences
Journal title:Proceedings of the National Academy of Sciences - PNAS, 2017-03, Vol.114 (12), p.3157-3162
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ASKA technology-based pull-down method reveals a suppressive effect of ASK1 on the inflammatory NOD-RIPK2 pathway in brown adipocytes
Author / Creator:Takayanagi, Saki ; Watanabe, Kengo ; Maruyama, Takeshi ; Ogawa, Motoyuki ; Morishita, Kazuhiro ; Soga, Mayumi ; Hatta, Tomohisa ; Natsume, Tohru ; Hirano, Tomoya ; Kagechika, Hiroyuki ; Hattori, Kazuki ; Naguro, Isao ; Ichijo, Hidenori
Publisher:London: Nature Publishing Group UK
Journal title:Scientific reports, 2021-11, Vol.11 (1), p.22009-22009, Article 22009
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The COX-2/PGE2 pathway suppresses apical elimination of RasV12-transformed cells from epithelia
Author / Creator:Sato, Nanami ; Yako, Yuta ; Maruyama, Takeshi ; Ishikawa, Susumu ; Kuromiya, Keisuke ; Tokuoka, Suzumi M. ; Kita, Yoshihiro ; Fujita, Yasuyuki
Publisher:London: Nature Publishing Group UK
Journal title:Communications biology, 2020-03, Vol.3 (1), Article 132
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Disruption of Sphingolipid Biosynthesis Blocks Phagocytosis of Candida albicans
Author / Creator:Tafesse, Fikadu G ; Rashidfarrokhi, Ali ; Schmidt, Florian I ; Freinkman, Elizaveta ; Dougan, Stephanie ; Dougan, Michael ; Esteban, Alexandre ; Maruyama, Takeshi ; Strijbis, Karin ; Ploegh, Hidde L
Publisher:United States: Public Library of Science
Journal title:PLoS pathogens, 2015-10, Vol.11 (10), p.e1005188-e1005188
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CHIP-dependent termination of MEKK2 regulates temporal ERK activation required for proper hyperosmotic response
Author / Creator:Maruyama, Takeshi ; Kadowaki, Hisae ; Okamoto, Noriaki ; Nagai, Atsushi ; Naguro, Isao ; Matsuzawa, Atsushi ; Shibuya, Hiroshi ; Tanaka, Keiji ; Murata, Shigeo ; Takeda, Kohsuke ; Nishitoh, Hideki ; Ichijo, Hidenori
Publisher:Chichester, UK: John Wiley & Sons, Ltd
Journal title:The EMBO journal, 2010-08, Vol.29 (15), p.2501-2514
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Na+/H+ exchanger isoform 3 expressed in apical membrane of gill mitochondrion-rich cells in Mozambique tilapia Oreochromis mossambicus
Author / Creator:Watanabe, Soichi ; Niida, Mayu ; Maruyama, Takeshi ; Kaneko, Toyoji
Publisher:Tokyo: Springer-Verlag
Journal title:Fisheries science, 2008-08, Vol.74 (4), p.813-821
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Histone H2B mutations in inner region affect ubiquitination, centromere function, silencing and chromosome segregation
Author / Creator:Maruyama, T ; Nakamura, T ; Hayashi, T ; Yanagida, M
Publisher:Chichester, UK: John Wiley & Sons, Ltd
Journal title:The EMBO journal, 2006-06, Vol.25 (11), p.2420-2431
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Epithelial recognition and elimination against aberrant cells
Author / Creator:Ayukawa, Shiyu ; Kamoshita, Nagisa ; Maruyama, Takeshi
Publisher:Berlin/Heidelberg: Springer Berlin Heidelberg
Journal title:Seminars in immunopathology, 2024-01, Vol.45 (4-6), p.521-532
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ArticlesPeer ReviewedFull Text
Publisher Correction: Epithelial recognition and elimination against aberrant cells
Author / Creator:Ayukawa, Shiyu ; Kamoshita, Nagisa ; Maruyama, Takeshi
Publisher:Berlin/Heidelberg: Springer Berlin Heidelberg
Journal title:Seminars in immunopathology, 2024-01, Vol.45 (4-6), p.551-554
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Increasing the efficiency of precise genome editing with CRISPR-Cas9 by inhibition of nonhomologous end joining
Author / Creator:Maruyama, Takeshi ; Dougan, Stephanie K ; Truttmann, Matthias C ; Bilate, Angelina M ; Ingram, Jessica R ; Ploegh, Hidde L
Publisher:New York: Nature Publishing Group US
Journal title:Nature biotechnology, 2015-05, Vol.33 (5), p.538-542
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Cell competition with normal epithelial cells promotes apical extrusion of transformed cells through metabolic changes
Author / Creator:Kon, Shunsuke ; Ishibashi, Kojiro ; Katoh, Hiroto ; Kitamoto, Sho ; Shirai, Takanobu ; Tanaka, Shinya ; Kajita, Mihoko ; Ishikawa, Susumu ; Yamauchi, Hajime ; Yako, Yuta ; Kamasaki, Tomoko ; Matsumoto, Tomohiro ; Watanabe, Hirotaka ; Egami, Riku ; Sasaki, Ayana ; Nishikawa, Atsuko ; Kameda, Ikumi ; Maruyama, Takeshi ; Narumi, Rika ; Morita, Tomoko ; Sasaki, Yoshiteru ; Enoki, Ryosuke ; Honma, Sato ; Imamura, Hiromi ; Oshima, Masanobu ; Soga, Tomoyoshi ; Miyazaki, Jun-ichi ; Duchen, Michael R. ; Nam, Jin-Min ; Onodera, Yasuhito ; Yoshioka, Shingo ; Kikuta, Junichi ; Ishii, Masaru ; Imajo, Masamichi ; Nishida, Eisuke ; Fujioka, Yoichiro ; Ohba, Yusuke ; Sato, Toshiro ; Fujita, Yasuyuki
Publisher:London: Nature Publishing Group UK
Journal title:Nature cell biology, 2017-05, Vol.19 (5), p.530-541
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The paxillin-plectin-EPLIN complex promotes apical elimination of RasV12-transformed cells by modulating HDAC6-regulated tubulin acetylation
Author / Creator:Kasai, Nobuhiro ; Kadeer, Ailijiang ; Kajita, Mihoko ; Saitoh, Sayaka ; Ishikawa, Susumu ; Maruyama, Takeshi ; Fujita, Yasuyuki
Publisher:London: Nature Publishing Group UK
Journal title:Scientific reports, 2018-02, Vol.8 (1), p.2097-10, Article 2097
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Rab5-regulated endocytosis plays a crucial role in apical extrusion of transformed cells
Author / Creator:Saitoh, Sayaka ; Maruyama, Takeshi ; Yako, Yuta ; Kajita, Mihoko ; Fujioka, Yoichiro ; Ohba, Yusuke ; Kasai, Nobuhiro ; Sugama, Natsu ; Kon, Shunsuke ; Ishikawa, Susumu ; Hayashi, Takashi ; Yamazaki, Tomohiro ; Tada, Masazumi ; Fujita, Yasuyuki
Publisher:United States: National Academy of Sciences
Journal title:Proceedings of the National Academy of Sciences - PNAS, 2017-03, Vol.114 (12), p.E2327-E2336
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Plectin is a novel regulator for apical extrusion of RasV12-transformed cells
Author / Creator:Kadeer, Ailijiang ; Maruyama, Takeshi ; Kajita, Mihoko ; Morita, Tomoko ; Sasaki, Ayana ; Ohoka, Atsuko ; Ishikawa, Susumu ; Ikegawa, Masaya ; Shimada, Takashi ; Fujita, Yasuyuki
Publisher:London: Nature Publishing Group UK
Journal title:Scientific reports, 2017-03, Vol.7 (1), p.44328-44328, Article 44328
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Inhibition of non-homologous end joining increases the efficiency of CRISPR/Cas9-mediated precise [TM: inserted] genome editing
Author / Creator:Maruyama, Takeshi ; Dougan, Stephanie K. ; Truttmann, Matthias ; Bilate, Angelina M. ; Ingram, Jessica R. ; Ploegh, Hidde L.
Journal title:Nature biotechnology, 2015-03, Vol.33 (5), p.538-542
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Erratum: Corrigendum: Increasing the efficiency of precise genome editing with CRISPR-Cas9 by inhibition of nonhomologous end joining
Author / Creator:Maruyama, Takeshi ; Dougan, Stephanie K ; Truttmann, Matthias C ; Bilate, Angelina M ; Ingram, Jessica R ; Ploegh, Hidde L
Journal title:Nature biotechnology, 2016-02, Vol.34 (2), p.210-210
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ArticlesPeer ReviewedFull Text
Corrigendum: Increasing the efficiency of precise genome editing with CRISPR-Cas9 by inhibition of nonhomologous end joining
Author / Creator:Maruyama, Takeshi ; Dougan, Stephanie K ; Truttmann, Matthias C ; Bilate, Angelina M ; Ingram, Jessica R ; Ploegh, Hidde L
Publisher:United States: Nature Publishing Group
Journal title:Nature biotechnology, 2016-02, Vol.34 (2), p.210-210
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ArticlesPeer ReviewedFull Text
Correction: Corrigendum: Increasing the efficiency of precise genome editing with CRISPR-Cas9 by inhibition of nonhomologous end joining
Author / Creator:Maruyama, Takeshi ; Dougan, Stephanie K ; Truttmann, Matthias C ; Bilate, Angelina M ; Ingram, Jessica R ; Ploegh, Hidde L
Publisher:New York: Nature Publishing Group US
Journal title:Nature biotechnology, 2016-02, Vol.34 (2), p.210-210
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Epithelial cells remove precancerous cells by cell competition via MHC class I–LILRB3 interaction

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Engineered red blood cells as carriers for systemic delivery of a wide array of functional probes

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Engineered erythrocytes covalently linked to antigenic peptides can protect against autoimmune disease

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ASKA technology-based pull-down method reveals a suppressive effect of ASK1 on the inflammatory NOD-RIPK2 pathway in brown adipocytes

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The COX-2/PGE2 pathway suppresses apical elimination of RasV12-transformed cells from epithelia

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Disruption of Sphingolipid Biosynthesis Blocks Phagocytosis of Candida albicans

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CHIP-dependent termination of MEKK2 regulates temporal ERK activation required for proper hyperosmotic response

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Na+/H+ exchanger isoform 3 expressed in apical membrane of gill mitochondrion-rich cells in Mozambique tilapia Oreochromis mossambicus

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Histone H2B mutations in inner region affect ubiquitination, centromere function, silencing and chromosome segregation

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Epithelial recognition and elimination against aberrant cells

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Publisher Correction: Epithelial recognition and elimination against aberrant cells

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Increasing the efficiency of precise genome editing with CRISPR-Cas9 by inhibition of nonhomologous end joining

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Cell competition with normal epithelial cells promotes apical extrusion of transformed cells through metabolic changes

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The paxillin-plectin-EPLIN complex promotes apical elimination of RasV12-transformed cells by modulating HDAC6-regulated tubulin acetylation

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Rab5-regulated endocytosis plays a crucial role in apical extrusion of transformed cells

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Plectin is a novel regulator for apical extrusion of RasV12-transformed cells

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Inhibition of non-homologous end joining increases the efficiency of CRISPR/Cas9-mediated precise [TM: inserted] genome editing

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Erratum: Corrigendum: Increasing the efficiency of precise genome editing with CRISPR-Cas9 by inhibition of nonhomologous end joining

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Corrigendum: Increasing the efficiency of precise genome editing with CRISPR-Cas9 by inhibition of nonhomologous end joining

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Correction: Corrigendum: Increasing the efficiency of precise genome editing with CRISPR-Cas9 by inhibition of nonhomologous end joining

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